||These observations suggest that some common factor may be
linking a number of high-risk phenotypes, in the absence of any changes in levels of
systolic, diastolic, or even pulse pressure. One possibility is that this antecedent is
related to activity of the renin-angiotensin-aldosterone system – many of these high
risk phenotypes are influenced by ACE inhibitors, including the non-modulating phenotype.
But once again the question as to whether insulin resistance is this common antecedent may
be worth considering.
Go to part II of this lecture
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