TREATMENT IDEA 1: Prevent PA from linking to its receptor on cells. Induce it to bind instead to decoys, such as soluble copies of the toxin receptor's PA binding site.

TREATMENT IDEA 2: Keep EF and LF from attaching to their binding sites on PA heptamers. Plug those sites with linked copies of a molecule that also has affinity for the sites.

TREATMENT IDEA 3: Block transport of EF and LF from the endosome into the cytosol by causing newly forming heptamers to incorporate a version of PA known as a dominant negative inhibitor (DNI). DNI-containing heptamers cannot move EF and LF accross the endosome's membrane.