prev next front |1 |2 |3 |4 |5 |6 |7 |8 |9 |10 |11 |12 |13 |14 |15 |16 |17 |18 |19 |20 |21 |22 |23 |24 |25 |26 |27 |28 |29 |30 |31 |32 |33 |34 |35 |36 |37 |38 |39 |review
Effects of VOO, TO, and OA treatments on total G protein αi2, αi3 and αq/11 subunits and PKCβ1 levels in aorta. SpragueDawley rats received vehicle (V), VOO (2 g·kg1), TO (1 g·kg1), or OA (1 g·kg1) p.o. every 12 h for 14 days. Insets show representative immunoblots of aortic G protein αi2 (A), G protein subunit αi3 (B), G protein subunit αq/11 subunit (C), and PLCβ1 (PLCβ1a + PLCβ1b) (D) after vehicle, VOO, TO, or OA administration. The amount of total protein loaded was 24 μg for Gαi2, αi3, and αq/11 subunits and 80 μg for PLCβ1. The columns represent the mean ± SEM (n = 5) of total protein levels quantified against standard curves and normalized to the protein content from vehicle-treated rats (taken as 100%). *, P < 0.05 and **, P < 0.01 vs. vehicle-treated rats (ANOVA).