James P. Fabisiak - Professional Activities
- 1991 - 1997 Small group workshops and problem based learning sessions for Medical Pharmacology, University of Pittsburgh
- 1991 - 1994 Cardiovascular/Pulmonary Pharmacology, University of Pittsburgh
- 1992 - 1994 Cell Culture Module in Physician Investigator Training Course, Department of Medicine, University of Pittsburgh
Seminars and Invited Lectureships
1990 Fabisiak, J.P., Evans, J.N., Absher, M. and Kelley, J. Differential production of and response to platelet-derived growth factor (PDGF) by rat and human lung fibroblasts. Presented at the Sixth International Colloquium on Pulmonary Fibrosis, Stowe, VT. 1990 Fabisiak, J.P., Evans, J.N., Gannon, D., Absher, M.P. and Kelley, J. Expression of platelet-derived growth factor A-chain (PDGF-A) mRNA in lung tissue and cells: Complex regulation during remodeling. Presented at Thomas L. Petty Aspen Lung Conference "Mechanisms of Lung Repair". 1992 Fabisiak, J.P. Expression of PDGF receptors on human lung fibroblasts and regulation by interleukin-1 b, (IL-1 b ), tumor necrosis factor- b (TNF- b ), and gamma-interferon ( g -IFN). Presented at the Seventh International Colloquium on Pulmonary Fibrosis, Cambridge, UK. 1998 Bifunctional anti/prooxidant role for metallothioneins: Redox regulation of copper binding. Magee Women's Research Institute, Pittsburgh, PA 1999 Mycoplasma, A “Steath” Pathogen? Lessons from Lung Transplantation. Dept. of Environmental & Occupational Health, University of Pittsburgh. 2001 Microbial & chemical stress interactions: Mycoplasma fermentans as a modulator of environmental disease. Div. Pulmonary, Allergy, Critical Care Medicine, University of Pittsburgh School of Medicine. 2004 Microbial & chemical stress interactions: Mycoplasma synergistically amplify lung fibroblast-derived cytokines in response to endogenous and environmental stimuli., Div. Of Pulmonary Medicine, Children's Hospital of Pittsburgh, Pittsburgh, PA. 2005 MAP kinase participation in amplified IL-6 release by human lung fibroblasts exposed to Ni and TLR-2 agonist, MALP-2. Poster presentation at Eight International Meeting on Mechanisms of Action of Inhaled Fibers, Particles, and Nanoparticles in Lung and Cardiovascular Disease. EPA Conference Center, Research Triangle Park, NC. 2008 Environmental and microbial stimuli interact to promote a proangiogenic phenotype in lung fibroblasts: Cooperation of prostanoid- and hypoxia-dependent signaling. Dept. Envir. & Occup. Health, University of Pittsburgh Graduate School of Public Health, Pittsburgh, PA.