A Developmental
Model of Children’s Early Conduct Problems:
Theory,
Validation, and Implications
Identification and Prevention of Early
Conduct Problems
Introduction
This
paper describes and applies a developmental model of early conduct problems
proposed earlier this decade. For a more
complete description of the model, please see the following papers.1, 2 This task will be carried out by reviewing
the model’s original tenets, examining its validity using two cohorts of
at-risk families, and offering recommendations for basic and applied studies.
The
rationale for developing a model beginning during infancy was based on the high
stability of antisocial behavior beginning in early childhood, the difficulty
of treating antisocial youth, and the disruption antisocial youth cause for
other individuals and society.3 Despite the plethora of research on
the treatment of antisocial behavior in childhood, efforts to prevent its development
have proven to be difficult. 4, 5 This
limited success may be due to our inability to fully understand either the
developmental trajectories leading to the disorder or the most appropriate
content and timing of the intervention.
As an example, past research on treatment of conduct problems has shown
that interventions implemented prior to school-age have a higher probability of
success.6 In response to the
need to more fully understand the origins of early conduct problems,7, 8
Shaw and Bell2 proposed a bridging model of early conduct problems
beginning in early childhood.
Initial
Bridging Model of Antisocial Behavior
The
goal of the bridging model was to integrate theory and normative empirical work
on young children’s development with studies of correlates of older children’s
conduct problems1 (CP). The
work of several investigators figured prominently in the development of the
bridging model. At a broad level,
Hirschi’s9 social control theory provided a mechanism from which to
understand parental influence, as the antisocial child’s lack of self-control
was postulated to emerge from his inability to form an attachment to parents in
early development. Sroufe’s10
conceptualization and application of attachment theory to early conduct
problems was also instrumental, describing how avoidant working models are
formed during infancy and demonstrating how they predisposed children to show
later noncompliant and hostile acting out behavior.11 Greenberg’s and Speltz’12
cognitive-affective model, also conceptualized from an attachment perspective,
provided specific examples of how parent-child interchanges from ages 2 to 4
would lead to early disruptive behavior based on the dyad’s inability to form a
goal-corrected partnership. Finally,
Patterson’s13 model of coercion applied principles from social
learning theory to explain how patterns of family interaction might produce
conduct problems in school-age children, a model adapted and validated in early
childhood by Martin.14
Martin’s work provided the critical empirical link for the model by
demonstrating longitudinal associations between unresponsive caregiving and coercive parent-child interaction, thereby
establishing a bridge between attachment and social learning models.
Methodologically,
we adopted the reciprocal effects model of Bell15 and the
transactional perspective of Sameroff.16 We also considered it critical to incorporate
the normal cognitive and emotional changes that children undergo from infancy
to school entry. Thus, our framework
considered (1) ongoing influences that parents and children have on each other,15 and (2) the previous behavior of both parents
and children in accounting for their later behavior16 within the context
of children’s rapidly evolving development.
As is evident from our use of several other earlier theoretical
frameworks, the model is an attempt to combine and integrate previous
perspectives on developmental psychopathology.
Its novelty rests primarily on its ability to synthesize perspectives
from disparate theoretical frameworks and different developmental periods to
provide a cohesive framework for understanding processes leading to the
antecedents of conduct problems in early childhood.
Integration of
Attachment and Social Learning Models
A
primary goal of the model was to account for sequencing of early disruptive
behaviors using a developmental framework.
One paradigm that has been used to understand the development of conduct
problems is coercion, a process that Patterson13 utilized to
describe the conflictual pattern of interaction
exhibited by disruptive school-age children and their families. In a coercive cycle, parent and child each
behave in a way that is aversive to the other in order
to control the other's behavior. As the
child becomes increasingly irritating, the parent escalates power assertion
techniques. As the child's aversive
behaviors increase in intensity and frequency, the parent acquiesces,
unwittingly reinforcing them. These
cycles eventually lead to the child's open defiance and behavior problems that
in later development include being away from home excessively, lying, stealing,
and more serious behaviors such as fire-setting. Patterson and colleagues8 formalized
the "early starter model," which is one of two pathways by which
children may emerge as chronically offending delinquent adolescents and
antisocial adults. The other pathway,
the late starter trajectory, emerges in early adolescence and in most cases has
been associated with less chronic and serious offending. According to Patterson’s early starter model,
families provide direct training in antisocial behavior for young boys through
their family management practices. While
Patterson13 acknowledges that children contribute to parent’s
ineffective parenting, greater emphasis is placed on parent than child
characteristics. Alternatively,
Moffitt’s17 early-starter model emphasizes impairments in child’s
early neuropsychological functioning, as manifested by the cognitive deficits
associated with ADHD (inattention, poor organization, and planning), which in
turn are postulated to elicit ineffective parent management strategies and a
trajectory of persistent conduct problems.
Attachment
theory has also been used to provide a framework for understanding the origins
of early conduct problems.18 Psychoanalysts such as Anna Freud and
Spitz were among the first to point out the importance of the social role
played by the mother in socioemotional
development. The mother's interaction
with the infant was seen as leading the infant to perceive her as accessible
and supportive, thus promoting ego development and movement through the
normative stages of socioemotional development. Ethological and evolutionary theory placed
still greater emphasis on the communicative function of emotions and social
relationships, as well as on the adaptive value of the mother-infant
relationship. Bowlby19
extended this theory by pointing out that signals such as smiles and cries
provide the foundation for attachment bonds that promote the infant's proximity
to protective adults and thus have survival value. Ainsworth's and Wittig’s20
differential theory, in turn, applied Bowlby's
general theory to individual differences.
She specified the characteristics of the caregiver, such as contingent
and appropriate responsiveness, that are presumed to produce secure versus
anxious attachments. Attachment security
is thought to reflect the infant’s internal working schema or model. This model lays the groundwork for patterns
of social information processing in early childhood, which in turn underlie the
child’s social and antisocial behavior.21 Securely attached infants would also be
expected to function more harmoniously with their mothers in compliance
situations because they are motivated to please the mothers, and attend to what
she approves or disapproves.22
Moving into the preschool period, Greenberg and Speltz12
contend that disruptive behavior is an attempt to get attention or control the
behavior of unresponsive or unpredictable caregivers in the absence of a
goal-corrected partnership. Accordingly,
secure preschoolers and their parents should be working together effectively in
household work and problem situations as the child becomes more adept at
managing his own emotions, and at enlisting the help of caregivers when
emotions threaten to become disorganizing.14
In
integrating attachment theory with Patterson’s coercion model, we believed it
was important to account for the quality of the parent-infant affective
relationship in influencing the course of social learning processes. Thus, infants who were shown less
contingently sensitive caregiving in the first 12
months would be more oppositional and defiant in responding to parental
requests for attention. The potential
for dyadic conflict was thought to peak between 18 and 30 months (i.e., the
terrible twos), when toddlers’ newfound mobility and potential for naive
mischief would stimulate parents to use control strategies and increase demands
for socially appropriate behavior. While
coercion theory posits that parental use of inconsistent, permissive, or overly
harsh discipline would reinforce children’s oppositional and aggressive
behavior, attachment theory suggests that in the first two years the quality of
the parent-child relationship would place specific dyads at greater risk for
engaging in these aversive interchanges, which in turn would set the stage for
escalating parent-child conflict and conduct problems at preschool-age.
Importantly,
several types of child attributes could potentially compromise the quality of
the parent-infant relationship, increase the likelihood of coercive parenting,
and ultimately, escalate the frequency and intensity of child disruptive
behavior. In addition to aggressive and
oppositional behavior, other forms of aversive child behavior include overactivity, emotional reactivity or unresponsivity,
or low behavioral inhibition, the latter expressed by an attraction to unknown
or even scary stimuli (e.g., electric plugs, high pitched sounds, walking
across streets). There is some evidence
to suggest that each of these factors may be directly related to later conduct
problems without considering parental influence. However, the effects of child factors are
likely mediated by the parent’s interpretation of their offspring’s behavior,
which is hypothesized to play a critical role in both the formation of
attachment bonds and the use of coercive discipline practices. It also is important to know how such aversive
child behaviors are influenced by parental intervention. For example, an infant whose fussiness
persists in the face of parental unresponsiveness or attempts to soothe the
infant’s discomfort would be expected to have more adverse outcomes than an
infant who desists from fussing on his/her own or in response to parental
comforting.
Influence of
Siblings
Patterson's13
model of coercion also links sibling interaction to the development of conduct
problems, hypothesizing that in homes in which family management practices were
disrupted, the additional stress of an older sibling would reinforce and
exacerbate the coercive style of the younger child. Data from Patterson’s 198423 study were consistent with the notion that siblings train
younger children to be coercive by modeling and then reinforcing aversive
behavior. However, this was a
cross-sectional study of school-age children; research on sibling influence of
early conduct problems is relatively limited.24
Contextual
Factors
The
model would be incomplete without integrating the influence of less proximal
contextual factors in the family’s environment.
Some theorists have suggested that the detrimental effects of family and
community risk factors should be mediated through more proximal interactions
parents have with young children.13, 2 Thus, the effects of such factors as parental
psychopathology, parental conflict, and social support may be at least
partially accounted for by the quality of the parent-child relationship.13,
2 Alternatively, there is evidence
to suggest that exposure to severe parental discord and neighborhood deviancy
may exert a direct influence on children’s CP.
Children exposed to such environments may show higher rates of CP
because it is modeled in the home and the neighborhood, or more indirectly
because it initially causes anxiety that may be channeled into aggressive and
oppositional behavior. For instance,
following exposure to a heated argument between parents, some children may have
a short fuse in interacting with family members and peers). Note these mechanisms are not mutually
exclusive. Research on parental adjustment
and intrafamily factors, such as parental depression,25 parental conflict,26 parenting
hassles,27 social support,28 and neighborhood
dangerousness29 all indicate significant associations with child
conduct problems. Particularly in
low-income contexts marked by sociodemographic risks
such as impoverished housing, high crime, and limited community resources, it
would be expected that the effects of within-family contextual factors would be
exacerbated by sociodemographic risk. For example, tolerance of deviant behavior in
the neighborhood may influence young children’s propensities to engage in
disruptive behavior.29 Just
as siblings reinforce power assertive methods parents model to resolve
parent-child and parent-parent conflict in the home, neighborhood peers,
particularly older ones, model coercive and conflictual
behavior in the neighborhood and thereby reinforce children’s tendencies to
show aggressive and hostile behavior within and outside of the family context.
In
summary, we concur with the observations of earlier investigators taking a
developmental perspective towards childhood psychopathology that early conduct
problems are generated as a result of transactions between children and their
environments over time.30, 31
Particularly during early childhood, it is important to take into
account transformations that occur in both child and parent behavior as the
child matures. For example,
temperamentally difficult infants might be more noncompliant as toddlers,
compared to easy infants. Moreover,
parents who are not responsive during infancy might be less involved and more
permissive with their toddlers. At the
same time, transactions between parent and child might help maintain
continuity: persistently noncompliant behavior makes enforcing rules more
difficult, and hostile parenting reinforces child aggression. In addition to child and parent behavior, it
is also necessary to consider the potential effects of stressors within and
outside the family that compromise the quality of the caregiving
environment. We now turn to examining
the model’s empirical validity.
Main Findings
Empirical
Validation of the Model
Data
from two sources have permitted us to test several of the model’s primary
tenets from infancy to the early school-age period.1 These include examining direct, additive, and
interactional effects of child, parenting, family,
and contextual factors on the emergence of early conduct problems (e.g., for
parenting, responsiveness during infancy and rejection during the toddler
period). Cohort I involves a sample of
100 children (60% boys), recruited prior to 12 months of age and followed until
school-age.32, 33 Cohort II is a sample of
310 boys and 55 girls recruited prior to 18 months of age and at present,
followed until age 8.34, 35
Because of funding constraints, follow-up has been more intensive for
boys than girls in Cohort II.
Participants
In
both cohorts, low-income mothers and their infants were recruited from the
Women, Infants, and Children (WIC) Nutritional Supplement Program of Allegheny
County, PA. WIC provides monetary
support for the purchase of nutritionally sound foods for low-income families
with children ages 0 to 5. At the time
of the infant's birth, mothers in Cohort I ranged in age from 17 to 36, 46%
were either married or living together, the majority of families were Caucasian
(61%) and the remainder were African-American
(39%). The mean family income in Cohort
I was approximately $14,000 per year with 72.5% of the families having yearly
earnings equal to or less than $12,000.
Sociodemographic characteristics in Cohort II
were comparable, but because families in this cohort were required to have an
additional sibling living at home, mother’s age was higher (i.e., x = 28
years, range = 17 and 43 years). In
addition, significantly more mothers in Cohort II were married or living
together (62% versus 46% in Cohort I).
Procedures
In
Cohort I, mothers were recruited at WIC offices when infants were between 6 and
11 months old and first seen in our laboratory at age one. Successive follow-ups for Cohort I occurred
in the lab at age 2, and at participants’ homes at
1.25, 5, and 7-8 years, and through the mail at age 3. Because of Cohort II’s
more intensive follow-up and larger sample size, for the purposes of the
present paper, a review of data from Cohort I will be limited to assessments
conducted in the child’s first three years.
In Cohort II, families were recruited at WIC sites when infants were
between 6 and 17 months old. A sub-sample of these
families (n = 65 boys and 55 girls) were initially seen in our lab at
age one. The remaining 245 boys were
initially seen at 1.5 years in the lab, with all boys successively assessed at
2 (lab and home visit), 3.5 (lab), 5 and 5.5 (home), 6 (lab), and 8 (home)
years of age. Follow-ups for girls
included assessments at age 1.5 and 2, with questionnaires completed by mail at
3.5 years. In both samples, family
members completed interactive tasks during laboratory and home assessments and
parents completed questionnaires on child and parental functioning and family
circumstances.
Testing the Model’s
Primary Tenets
A
couple of issues are important to note before proceeding to a discussion of our
results. First, in all cases
experimenter-wise error was controlled for in testing associations to ensure results
were not attributable to chance. The
reader is referred to the specific journal articles in which each finding was
originally reported for more details.
Second, to date, nearly all of our results are based on measures of
conduct problems that tap a heterogeneous constellation of symptom patterns,
including defiant, aggressive, impulsive, and hyperactive behaviors. We have not considered, for example, specific
subtypes of children with co-occurring patterns of ADHD and/or
oppositional/conduct problems or children who show covert versus overt conduct
problems. There are exceptions to this
trend;35, 36 however, we are
still in the process of examining the antecedents of other specific patterns of
conduct problems, including children with co-occurring externalizing and
internalizing problems and those who show predominantly overt versus covert
antisocial behavior. This represents a
limitation of the research and its support of the model, which is primarily
directed at uncovering antecedents of early onset conduct problems,
characterized by high emotional reactivity and a pattern of overt and
(eventually) covert antisocial activity.
Maternal Responsivity and Conduct Problems
Using
these two samples of low-income boys, we have been able to test the predictive
validity of several of the model’s primary hypotheses. A primary goal was to validate parental and
child contributors of early conduct problems beginning in infancy. With regard to parental influences, the
model’s focus during the first year has been on maternal unresponsiveness. In accord with attachment theory19
and coercion theory,13 we postulated that a
lack of sensitivity to the infant during the end of the first year would be related
to later coercive exchanges between parents and children and ultimately to
higher rates of children’s conduct problems.
This issue has been investigated using two different
observationally-based methodologies. The
first strategy was to measure the mother’s contingent level of responsiveness
in relation to infant bids for attention.
Using Martin’s14 high-chair procedure, in which one-year olds
are placed in a high-chair with nothing to do while mothers are instructed to
complete a questionnaire and attend to the infant, maternal
unresponsiveness to the infant’s bids for attention was related to observed
noncompliant and/or aggressive behavior at age 2 and maternal report of conduct
problems at ages 3 to 3.5 in both cohorts.32, 34 Relations between responsiveness and maternal
reports of CBCL Externalizing problems at 24 and 42 months are displayed for
Cohort II in Figure I below. This
replicates the work of Martin,14 who found
maternal unresponsiveness associated with noncompliance at age 2 and coercive
child behavior at 3.5. It is also
consistent with the findings of Wakschlag and Hans,37 who found an association between maternal
unresponsiveness during infancy and later conduct problems. Interestingly, in both of our studies and
Martin’s, these relations were valid only for boys. While both of our cohorts involve low-income
boys, Martin’s used a middle-class sample, suggesting generalizability
of the findings to lower risk populations of boys. The issue of sex differences is addressed
later in the paper.
Insert Figure 1
about here
We
also examined the construct of maternal responsivity
using the Strange Situation. The infant’s
behavior during the Strange Situation is believed to reflect the quality of the
caregiver’s contingent sensitivity with the infant. Consistent with studies of high-risk samples,11, 38, 39 relations were found
between infant attachment insecurity at 1 and 1.5 years and conduct problems
when children were age 340 and age 5.41 The magnitude of relations between infant
attachment status and later conduct problems was stronger at age 5 than at age
3, particularly for infants with the disorganized pattern of attachment. This finding is consistent with a recent
meta-analysis of the relation between the disorganized pattern and later
conduct problems.42
Infant
Characteristics, Parenting, and Conduct Problems
Another
primary component of the model is infant characteristics, particularly
behaviors that would be directly linked to later disruptive behavior or be
viewed as aversive by parents, thereby evoking coercive interchanges and
escalating levels of conduct problems.14, 17
Findings from both Cohorts I and II reveal direct
and interactive effects of observed infant characteristics on later conduct
problems. Infant persistence, a measure
of how often the infant makes initial bids for attention and continues
to fuss in relation to the mother’s unresponsiveness, was related to observed
aggression at age 2 in Cohort I and maternal report of conduct problems at age
3.5 in Cohort II (see Figure 1). In both
cases, these relations were significant only for boys. Finally, in both Cohorts, observed aggression
and noncompliance at age 2 have been significantly associated with maternal
report of conduct problems at ages 3 to 3.5 (Figure 1).32, 34
The
model also postulates that the interaction of parent and infant characteristics
should add unique variance to the prediction of early conduct problems after
accounting for each factor’s direct effects.
Evidence also supports this supposition.
In Cohort I, the interaction between maternal responsiveness at age 1
and observed aggression at age 2 contributed additional variance to maternal
report of conduct problems at age 3.32 In Cohort II, the interaction between high
infant persistence and low maternal responsiveness on the high-chair task also
contributed unique variance to maternal report of age 3.5 conduct problems
after accounting for each factor’s direct effects.34 In both cases, more aversive child behavior
coupled with unresponsive parenting appeared to heighten risk for later
outcome, and in both cases the interaction was valid only for boys. A similar approach was applied to our
findings regarding infant attachment status.
In Cohort I, there was a strong association between the disorganized
classification during infancy and preschool conduct problems (i.e., 6 out of 10
disorganized infants attained clinical-level symptomatology
on the CBCL Aggression factor at age 5).
When maternal perception of infant difficultness was examined in
interaction with attachment security, prediction of risk status was further
improved. Among mothers who rated their
infant as being above the median in difficulty and who had infants with a
disorganized attachment classification, rates of clinically-significant aggressivity were 100% (6 out of 6 cases), whereas those
with disorganized attachments and below-median perceived difficulty were all
below clinical threshold on aggression.41
Moving
from the first to second year, the model’s emphasis shifts to how parents
respond to the infant’s increase in mobility and expression of anger. Unfortunately for parents, toddling is
accompanied by an increased desire to “own” most toys the infant comes in
contact with (i.e., ‘mine’), evoking frustration and expression of anger when
these desires are not met. In addition
to responding appropriately to the infant’s disruptive behavior, the parent
must also set limits to protect the infant, other family members, pets, and
valuable/dangerous objects from the infant’s limited cognitive understanding of
such concepts as gravity, electricity, and differentiation of living versus
nonliving organisms. Thus, a primary
objective has been to assess parent’s ability to maintain a positive and nonhostile approach to these shaping and ‘coaching’ tasks
during this challenging period. For both
boys and girls, children whose parents were observed to be rejecting at age 2
during a laboratory-based clean-up task demonstrated a heightened risk for
conduct problems at age 3.5 (see Figure 1).34 Furthermore, a composite score of observed
rejecting parenting at ages 1.5 and 2 differentiated clinically-significant
levels of boys’ conduct problems at ages 5.5-6 and 8 according to both
parent and teacher reports.36
These findings are consistent with previous studies on the effects of
rejecting or overcontrolling parenting conducted with
preschool-age children,43 school-age children and adolescents,44,
45 and the model’s emphasis on the significance of parenting practices
during the toddler period.
Coercion and
Intra-Family Conflict
Another
primary goal has been to trace the spread of coercive and conflictual
relationships within families to relationships in other contexts. According to Patterson,13
coercive interactions should extend from the parent-child relationship to the
behavior of siblings, then generalize to other adult-child and peer
relationships outside of the home. We
would add to this the significance of interparental
conflict, which may provide modeling of conflict resolution strategies above
and beyond the ‘direct’ training children receive from parents and siblings. In
support of the spread-of-coercion hypothesis, interparental
conflict at ages 2 and 3.5 and parent-child and inter-sibling conflict assessed
at age 5 were related to parent-child conflict at age 5, and teacher-child and
peer conflict at age 6. Both additive
and interactive effects were found for these dyadic predictor variables
according to both parental and teacher reports.46 Patterson also suggests that older
siblings help in the “training” of younger sibling’s aggression. In support of this hypothesis, we found
prolonged and aggressive sibling conflict to be associated with maternal report
of aggressive child behavior at ages 5 and 6, after accounting for the
influence of the target child’s early externalizing symptoms and rejecting
parenting. Interactive effects of
rejecting parenting, assessed at age 2, and sibling conflict assessed at age 5,
were also found for both parent and teacher reports of age 6 aggressive
behavior at school after accounting for main effects,35 the results
of which are displayed in Figures 2A and 2B below. According to both parental and teacher
report, in cases where parental rejection at age 2 and sibling conflict at age
5 were high, reports of child aggressive behavior were elevated. These findings suggest that in addition to
direct relations between rejecting parenting, early disruptive behavior,
sibling conflict, and conduct problems at school entry, the presence of more
than one of these risk factors further increases children’s vulnerability for
conduct problems at home and at school.
Insert Figures
2A and 2B about here
Early
Predictors of School-Age Conduct Problems
A
further test of the model’s validity is to examine if early caregiving
and contextual factors that compromise the quality of caregiving
differentiate clinically-meaningful conduct problems across contexts during the
school-age period. We employ a person-oriented
approach for these analyses to trace the differential pathways of individuals
sharing common risk factors or a common outcome.
In
the first study of this type, we identified groups of families who shared
characteristics across four domains: child characteristics, maternal parenting
behavior, family context, and sociodemographic characteristics.47
We then examined how such groups of children fared on measures of
conduct problems according to both parents and teachers at school-age. Importantly, this study compared findings from
our community sample of low-income, ethnically diverse infants with those of
Susan Campbell’s predominantly middle-class, European American (EA)
preschoolers identified on the basis of ADHD symptomatology. In both samples, the children who showed the
most consistent pattern of conduct problems at school-age (age 6 in the Shaw
sample, age 9 in the Campbell sample) were marked by risk across child, parent,
family, and sociodemographic domains. At the time of the study entry (age 1.5 in
the Shaw sample, age 3 to 4 in the Campbell sample), in both cohorts the
multiple risk group demonstrated elevated hyperactivity and aggression, more
negative and less positive parenting, and higher rates of maternal depressive symptoms,
stressful life events, and sociodemographic risk than
families in clusters with fewer risk factors (e.g., no-risk group,
child-risk-only group). In both samples,
boys in the multi-problem group were observed to show the most disruptive
behavior at study entry and display more conduct problems and lower social
competence at follow-up relative to other risk groups.
To
test the validity of early starter models proposed by Moffitt48 and
Patterson,13 we identified
clinically-meaningful cases at school-age and looked back at factors that
discriminated group status in early childhood.36 The Kiddie-Schedule
for Affective Disorders - Epidemiologic Version (K-SADS-E)49 was
administered to mothers about their 8 year-old sons, from which diagnoses of
DSM-IV disruptive disorders were derived.
Teachers completed the Teacher Report Form50 at age 8, from
which scores greater than or equal to the 90th percentile on the
Aggression factor were used to establish clinical impairment. This cutoff score was chosen to ensure that
children in the clinical group were qualitatively and clinically distinct from
their peers, but permitted a sufficiently large sub-sample of impaired children
to conduct comparative analyses.
Children who met criterion for Oppositional Defiant Disorder (ODD),
Conduct Disorder (CD), or ODD or CD and Attention Deficit Hyperactivity
Disorder (ADHD) at age 8 according to K-SADS interviews were marked by early
problem behavior and multiple family risk factors (e.g., maternal depression,
low social support, rejecting parenting) that were evident in the second year
of life. However, because many of the
measures of early child and family functioning were derived from maternal
report, the sole exception being observed rejecting parenting and quality of
the home environment (i.e., the HOME Inventory), it was important to
corroborate the results using teacher reports.
Maternal reports of infant negative emotionality and age 2 externalizing
problems were not related to clinically-meaningful conduct problems at
school-age as rated by teachers.
However, teacher-identified aggressive children were more likely to live
in families characterized by maternal depression, neighborhood dangerousness,
low social support, and impaired parenting when children were 1.5 to 2 years
old.
Finally,
we applied a semiparametric mixture model51
to examine developmental trajectories of overt CP from ages 2 to 8, including
assessments at ages 3.5, 5, and 6.52
This technique permits the identification of groups of individuals who
share similar patterns of behavior over time, and to identify risk factors that
differentiate trajectories. Consistent with research on older children,53 four developmental trajectories were
identified: a persistent problem trajectory, a moderate-level desister trajectory, a low-level desister
trajectory, and a persistent low trajectory.
Risk factors evaluated when children were between ages 1.5 and 2
included child behavioral inhibition, maternal
depressive symptoms, and rejecting parenting, all three of which discriminated
the developmental trajectories of children in the persistent high group from
those in the persistent low group even after the effects of other risk factors
were accounted for in the analysis.
Children who were less inhibited, whose mothers reported higher rates of
depressive symptoms and showed higher rates of rejecting parenting were more
likely to be in the chronic group.
All
three sets of person-oriented analyses suggest that young children’s pathways leading
to serious conduct problems across context are marked by multiple risk
factors across domains rather than merely the perception of the child as
difficult or behaviorally disruptive during infancy. These findings have implications for
interventionists and preventionists interested in
identifying target populations during early childhood. From our findings and those of others
studying the early antecedents of conduct problems,54,
39 it is clear that the development of antisocial behavior in children is
embedded within a context of biological characteristics of the child and
caregiver, developmental history, and community disruption.55 To be effective, interventions will need to
address the ecology within which the most serious forms of antisocial behavior
emerge. However, one must take seriously
the reservations of others who have tried to intervene with high-risk families
and failed because they neglected to address contextual factors.56 Salvador Minuchin,
the founder of structural family therapy, gave up working with low-income,
high-risk families because he concluded it was analogous to putting band-aids
on people who require surgery.57
Interventions that adopt the principles of multi-systemic therapy,58
which address both within-family (e.g., parent-child relations, parental
support and adjustment) and extra-familial issues (neighborhood safety,
accessibility to resources), are recommended.
It also is imperative that interventions are targeted to the
developmental challenges of early childhood and tailored to the demands of the
environments in which the child operates, including coordination of ‘parenting’
techniques with day care or preschool personnel. This latter approach is consistent with the
recent work of Webster-Stratton.59 In working with Head Start samples,
her interventions have been coordinated to ensure parents and teachers employ
similar techniques across setting.
Overall
there is broad support for the validity of the components of the model that
have been tested. We have found that
from ages 1 to 8, child and parenting variables contribute additively and often
interactively to an escalation in child conduct problems, distant and rejecting
parenting, and coercive parent-child relationships. Parental and sibling conflict appear to
exacerbate child and parenting risk factors in an additive and interactive
manner, reinforcing patterns of disruptive behavior and conflictual
relations with adults and peers.
Discussion
Applying
a Developmental Perspective
Implicit
in the model is an integration of developmental processes to the study of child
psychopathology. Part of the marriage
between developmental psychology and psychopathology has involved taking advantage
of normative milestones of childhood to capture points of transition.47 The FAST Track Project4 is a prime
example of this approach, in which the transition to full-day schooling was
used to study the onset and prevention of serious conduct problems. Several prospective studies have also been
initiated in early childhood to take advantage of transitions in the
parent-child relationship.60
Still, there remains a dearth of treatment and prevention studies
initiated in early childhood that have been guided by developmental
theory. Interventions that capitalize on
the challenges of parental responsiveness in the first year, and the
integration of sensitive caregiving with firm, but
not hostile, parenting in the second and third years are recommended. In addition and as noted above, it behooves
treatment researchers to consider the impact of all family members and to test
their approach with families characterized by risk factors across child,
parenting, family, and sociodemographic domains. Our research and that of several other
investigative teams61, 47, 62, 63 suggest that children from
multi-problem contexts pose the greatest likelihood of early onset and
maintenance of psychopathology, particularly conduct problems.
There
is also a need for developmentally-sensitive studies initiated in early
childhood that incorporate behavior genetic designs. Without a genetic design it is not possible
to tease apart genetic from environmental influence. In particular, adoption studies have the
potential to uncover evocative gene-environment correlations between heritable
child characteristics (e.g., impulsivity, activity) and environmental responses
(e.g., rejecting parenting).
Additionally, they can identify gene x environment interactions which
involve the potential moderating effects of the environment in relation to
genetic risk for problem behavior (e.g., parenting of adoptive parents may
increase or decrease the risk of infant negative emotionality).64
Another
concern is our lack of understanding of the development of conduct problem
among girls. Girls are less likely to
show serious conduct problems compared to boys in middle childhood and
adolescence; however, sex differences do not emerge consistently until age 4 or
5.65, 66 Several hypotheses have
been offered to account for girls’ more rapidly decreasing rates of
oppositional and aggressive behavior from ages 2 to 5 (e.g., socialization
pressures for girls to be more compliant and less physically aggressive), but
longitudinal research is needed to test these suppositions. We also lack knowledge about the
developmental trajectories of girls who continue to show clinically-significant
rates of conduct problems throughout the preschool and school-age periods. In following our own two cohorts, we have
been struck by sex differences in the magnitude of relations between early
child and parenting risk factors and later conduct problems. Boys consistently exhibit greater
vulnerability to the effects of environmental adversity (e.g., parental
unresponsiveness) and infant negative emotionality, findings that also have
been replicated by Martin.14 These results are consistent with sex
differences found for neuropsychological disorders of early childhood such as
ADHD, learning disabilities, and autism, for which boys outnumber girls by a
wide margin.66
A
similar lack of knowledge is available on the effects of ethnicity. In
following our two cohorts over the past decade, the disparity in risk factors
continues to impress upon us the need for more research and attention to the
lack of resources within urban, minority communities. In our longitudinal studies, this inequality has been
most evident among African American (AA) families in comparison to European
American families. A large percentage of
our AA families live in segregated housing projects marked by poor housing,
high crime rates, low accessibility to resources (e.g., shopping,
transportation, medical care) and employment opportunities. Thus, neighborhood quality and ethnicity are
confounded because of the over-representation of AA families living in
impoverished conditions. Nonetheless, it
is worth discussing the issue of ethnicity separately because of idiosyncratic
issues associated with minority status.67
It
is only recently that researchers have begun to seriously consider the effects
of ethnicity on child conduct problems substantively, rather than as a factor
to be “controlled.” In some studies, sociodemographic factors have been found to account for
differences in antisocial behavior between European American (EA) and AA
families;68 however, in other cases
differences persist.69, 29
For example, ethnic differences in both relevant child and parenting
characteristics (e.g., hostile attributional bias,
authoritarian parenting style) have been identified among AA families,29, 70 but not always linked to higher
rates of CP particularly in low-income, urban samples.
Summary and Conclusions
This
paper has described a developmental model of early conduct problems and
reported findings about its validity with two samples of high-risk
children. Major tenets of the model have
been confirmed and advance our understanding of the processes by which early
parent and child characteristics and other familial and extra-familial factors
influence the development of child conduct problems at school entry. In terms of the implications of our
findings for prevention, it is clear that children with risk factors in
multiple domains face the greatest risk of becoming persistent early starters.
Thus, successful interventions with these families will need to be
comprehensive and tailored to the issues that compromise individual parent’s
abilities to provide safe and caring environments for their offspring.
References
1. Shaw DS, Bell RQ, Gilliom
M. A truly early starter model of
antisocial behavior revisited. Clinical Child and Family Psychology Review, 2000; 3:
155-172.
2. Shaw DS, Bell RQ. Developmental theories of
parental contributors to antisocial behavior. Journal of Abnormal
Child Psychology. 1993; 21: 493-518.
3. Loeber R, Stoughamer-Loeber M.
Development of juvenile aggression and violence: Some common
misconceptions and controversies. American Psychologist. 1998; 53:
242-259.
4. Conduct Problems Prevention Research
Group. A developmental and clinical
model for the prevention of Conduct Disorder: The FAST Track Program. Development and
Psychopathology. 1992; 4: 509-527.
5. Reid J.
Prevention of conduct disorder before and after school entry: Relating
interventions to developmental findings.
Development and Psychopathology. 1993; 5: 243-262.
6. Dishion T, Patterson GR. Age effects in parent training outcome. Behavior Therapist.
1992; 23: 719-729.
7. Moffitt TE, Caspi
A, Dickson N, Silva P, Stanton W.
Childhood-onset versus adolescent-onset antisocial conduct problems in
males: Natural history from ages 3 to 18 years. Development and
Psychopathology. 1996; 8: 399-424.
8. Patterson, GR, Capaldi
D, Bank L. An early starter model for
predicting delinquency. In D Pepler, & RK Rubin (Eds.), The
development and treatment of childhood aggression. 1991;
9. Hirschi T. Causes of delinquency. 1969;
10. Sroufe LA.
Infant-caregiver attachment and patterns of adaptation in pre-school: The roots
of maladaptation and competence. In M Perlmutter
(Ed.),
11. Erickson MF, Sroufe
LA, Egeland B. The relationship
between quality of attachment and behavior problems in preschool in a high-risk
sample. In I Bretherton
& E Waters (Eds.), Growing points of attachment theory and research. Monographs of the
Society for Research in Child Development. 1985; 50, Nos. 1-2,
147-167.
12. Greenberg MT, Speltz
ML. Contributions of attachment theory
to the understanding of conduct problems during the preschool years. In J Belsky
& T Negworski (Eds.), Clinical implications of
attachment (pp. 177-218). 1988; Hillsdale, New Jersey: Earlbaum.
13. Patterson GR.
A social learning approach: 3. Coercive family process. 1982;
Eugene, OR: Castalia.
14. Martin J.
A longitudinal study of the consequences of early mother-infant interaction:
A microanalytic approach. Monographs of the
Society for Research in Child Development. 1981;
46.
15. Bell RQ.
A reinterpretation of the direction of effects in
studies of socialization. Psychological Review. 1968: 75: 81-95.
16. Sameroff AJ. Prevention of developmental
psychopathology using the transactional model: Perspectives on host, risk
agent, and environment interactions.
Paper presented at the Conference on the Present Status and Future Needs
of Research on Prevention of Mental Disorders. 1990;
17. Moffitt TE.
Adolescence-limited and life-course-persistent antisocial behavior: A
developmental taxonomy. Psychological Review. 1993; 100:
674-701.
18. Greenberg MT, Speltz
ML, DeKlyen M.
The role of attachment in the early development of
disruptive behavior problems. Development and Psychopathology.
1993; 5: 191-213.
19. Bowlby J. Attachment.
1969; New York: Basic Books.
20. Ainsworth MDS., Wittig D. Attachment and exploratory
behavior of one-year-olds in a strange situation. In BM Foss (Ed.), Determinants of
infant behavior (Vol. 4). 1969; London: Metheun.
21. Sroufe LA, Fleeson J. Attachment and the construction of relationships. In W Hartup
& Z Rubin (Eds.), Relationships and development. 1986;
Hillsdale, NJ: Erlbaum.
22. Stayton DJ, Hogan R, Ainsworth MDS. Infant obedience and maternal behavior: the
origins of socialization reconsidered. Child Development. 1971; 42:
1057-1069.
23. Patterson
GR, Dishion TJ, Bank L. Family interaction: A process model of
deviancy training. Aggressive
Behavior. 1984; 10: 253-267.
24. Volling BL, Belsky J. The
contribution of mother-child and father-child relationships to the quality of
sibling interaction: A longitudinal study.
Child Development. 1992;
63: 1209-1222.
25. Zahn‑Waxler
C, Iannotti RJ, Cummings EM, Denham S. Antecedents of problem
behaviors in children of depressed mothers. Development and
Psychopathology. 1990; 2: 271‑292.
26. Jouriles EN, Murphy
CM, Farris AM, Smith DA, Richters JE, Waters E.
Marital adjustment, parental disagreements about child rearing, and behavior
problems in boys: Increasing the specificity of the marital assessment. Child Development. 1991; 62: 1424-1433.
27. Crnic KA,
Greenberg, MT. Minor parenting stresses with
young children. Child
Development. 1990; 61: 1628-1637.
28. Crnic KA, Greenberg
MT, Ragozin AS, Robinson NM, Basham RB. Effects of stress and
social support on mothers and premature and full-term infants. Child Development.
1983; 57: 209-217.
29. Winslow EB, Shaw
DS. Relations
between early parenting and child externalizing behavior in families from
different sociodemographic backgrounds. Presented at the convention
of the International Society for Infant Studies. 1996;
30. Sameroff AJ,
31. Thomas A, Chess S, Birch H. Temperament and behavior
disorders in children. 1968.
New York: New York University Press.
32. Shaw DS, Keenan K, Vondra
JI. Developmental precursors of
externalizing behavior: Ages 1 to 3. Developmental Psychology. 1994; 30:
355-364.
33. Vondra JI, Shaw DS, Chrisman J, Cohen L, Swearinger E, Owens EB. Continuity and change in attachment across
the second year of life. Development and Psychopathology. In
press.
34. Shaw DS, Winslow EB, Owens EB, Vondra JI, Cohn JF, Bell RQ. The development of early externalizing
problems among children from low-income families: A transformational
perspective. Journal
of Abnormal Child Psychology. 1998; 26: 95-107.
35. Garcia M, Shaw DS, Winslow EB, Yaggi K. Destructive
sibling conflict and the development of conduct problems in young boys. Developmental Psychology.
2000; 36: 44-53.
36. Shaw DS, Garcia M, Winslow EB, Owens EB. Pathways leading to
school-age externalizing disorders.
Presented at the Conference of the Life History Research
Society. 1999;
37. Wakschlag. L, Hans
S. Relation of
maternal responsiveness during infancy and the development of behavior problems
in high-risk youths. Developmental Psychology. 1999; 35:
569-579.
38. Lyons‑Ruth K, Alpern
L, Repacholi B.
Disorganized infant attachment classification and
maternal psychosocial problems as predictors of hostile‑aggressive
behavior in the preschool classroom.
Child Development.
1993; 64: 572‑585.
39. Renken B, Egeland B, Marvinney D, Mangelsdorf S, Sroufe A. Early childhood antecedents
of aggression and passive-withdrawal in early elementary school. Journal of Personality.
1989; 57: 257-281.
40. Shaw DS, Vondra
JI. Attachment security and maternal
predictors of early behavior problems: A longitudinal study of low-income
families. Journal
of Abnormal Child Psychology. 1995: 23: 335-356.
41. Shaw DS, Owens EB, Vondra
JI, Keenan K, Winslow EB. Early risk factors and pathways in the development of early
disruptive behavior problems. Development and Psychopathology. 1996;
8: 679-699.
42. van Ijzendoorn MH, Schuengel C, Bakermans-Kranenburg
MJ. Disorganized attachment in early
childhood: Meta-analysis of precursors, concomitant, and sequelae.
Development and Psychopathology. 1999; 11: 225-250.
43. Campbell SB, Pierce EW, Moore G, Marakovitz S, Newby K.
Boys' externalizing problems at elementary school: Pathways from early
behavior problems, maternal control, and family stress. Development and
Psychopathology. 1996; 8: 701-720.
44. Loeber R, Dishion TJ. Early
predictors of male delinquency: A review.
Psychological Bulletin. 1983; 94: 68-99.
45. McCord W, McCord J, Zola IK. Origins of crime.
1959; New York: Columbia University Press.
46. Ingoldsby E, Shaw
DS, Garcia M. Intra-familial
conflict in relation to boys’ adjustment at school. Development and
Psychopathology. In press.
47. Campbell SB, Shaw DS, Gilliom
M. Early externalizing
behavior problems: Toddlers and preschoolers at risk for later maladjustment. Development and Psychopathology.
2000; 12: 467-488.
48. Moffitt T.
Juvenile delinquency and attention deficit disorder: Boys’ developmental
trajectories from age 3 to 15. Child Development. 1990; 61:
893-910.
49. Puig-Antich J, Orvaschel H, Tabrizi MA, Chambers
W. The Schedule for Affective Disorders and Schizophrenia for School-age
Children-Epidemiologic Version ed 3. 1980; New
York: New York State Psychiatric Institute and Yale University School of
Medicine.
50. Achenbach
TM, Edelbrock C. Manual for the Teacher’s Report Form
and Teacher Version of the Child Behavior Profile. 1986; Burlington:
University of Vermont Department of Psychiatry
51. Nagin DS. Analyzing developmental
trajectories: A semiparametric group-based approach. Psychological Methods.
1999; 4: 139-157.
52.
Shaw DS, Gilliom
M, Ingoldsby EM, Schonberg MA. Developmental
Trajectories in Antisocial Behavior from Infancy to Adolescence. Presented at the Biennial
Meeting of the Society for Research in Child Development. 2001;
53. Nagin DS, Tremblay
RE. Trajectories of
boys’ physical aggression, opposition, and hyperactivity on the path to
physically violent and nonviolent juvenile delinquency. Child Development.
1999; 70: 1181-1196.
54. Campbell SB.
Hard-to-manage preschool boys: Externalizing behavior, social
competence, and family context at two-year-follow-up. Journal of Abnormal
Child Psychology. 1994; 22: 147-166.
55. Dishion TJ, French DC, Patterson GR. The development and ecology
of antisocial behavior. In D Cicchetti & DJ Cohen (Eds), Developmental
psychopathology: Vol 2. Risk, disorder, and
adaptation (pp. 421-471).
1995; New York: Wiley.
56. Kazdin AE. Conduct disorders in childhood and
adolescence (2nd Edition). 1995; Newbury Park, CA: Sage.
57. Malcolm J. The one-way
mirror. New
Yorker. 1979.
58. Henggeler SW, Bourdin CM. Family
therapy and beyond: A multisystemic approach to
treating the behavior problems of children and adolescents. 1990; Pacific
Grove, CA: Brooks/Cole.
59. Webster-Stratton C. Preventing conduct
problems in head start children: strengthening parenting competencies. Journal of Consulting
and Clinical Psychology. 1998; 66: 715-730.
60. Belsky J, Woodworth
S, Crnic K.
Trouble in the second year: Three questions about family
interaction. Child
Development. 1996; 67: 556-578.
61. Ackerman BP, Izard CE, Schoff
K, Youngstrom EA, Kogos
J. Contextual risk caregiver emotionality, and the problem behaviors of six- and
seven-year-old children from economically disadvantaged families. Child Development.
1999; 70: 1415-1427.
62. Rutter M, Cox A, Tupling C, Berger M, Yule W. Attainment and adjustment in two geographical
areas: 1. The
prevalence of psychiatric disorder. British Journal of Psychiatry. 1975;
126: 493-509.
63. Sameroff AJ, Seifer R, Bartko T. Environmental perspective
on adaptation during childhood and adolescence. In SS Luthar,
JA Burack, D Cicchetti,
J.R. Weisz (Eds.), Developmental
Psychopathology (pp. 507-526). 1997; Cambridge, UK: Cambridge
University Press.
64. O’Connor TG, Deater-Deckard
K, Fulker D, Rutter M., Plomin R.
Genotype-environment correlates in late childhood and early adolescence:
Antisocial behavioral problems and coercive parenting. Developmental Psychology.
1998; 34: 970-981.
65.
Achenbach TM. Manual
for the Child Behavior Checklist 2/3 and 1992 profile. 1992;
Burlington: University of Vermont Department of Psychiatry.
66. Keenan K, Shaw DS. Developmental influences on young girls'
behavioral and emotional problems. Psychological Bulletin. 1997; 121:
95-113.
67. Wilson WJ. When work
disappears: The world of the new urban poor. 1996; NY: Alfred A.
Knopf.
68. Hinshaw SP, Park
T. Research problems and issues: Toward
a more definitive science of disruptive behavior disorders. In HC Quay, AE Hogan (Eds), Handbook of disruptive behavior disorders.
In press; New York: Plenum Press.
69. Dodge KA, Pettit GS, Bates JE, Valente E. Social
information-processing patterns partially mediate the effect of early physical
abuse on later conduct problems. Journal of Abnormal Psychology. 1995;
104: 632-643.
70. Deater-Deckard K,
Dodge KA, Bates JE, Pettit GS. Physical
discipline among African American and European American mothers: Links to
children’s externalizing behaviors. Developmental Psychology.
1996; 32: 1065-1072.